Search results for "energy homeostasis"

showing 10 items of 44 documents

The corticotrophin-releasing factor/urocortin system regulates white fat browning in mice through paracrine mechanisms.

2015

Objectives:\ud The corticotrophin-releasing factor (CRF)/urocortin system is expressed in the adipose tissue of mammals, but its functional role in this tissue remains unknown.\ud \ud Methods:\ud Pharmacological manipulation of the activity of CRF receptors, CRF1 and CRF2, was performed in 3T3L1 white pre-adipocytes and T37i brown pre-adipocytes during in vitro differentiation. The expression of genes of the CRF/urocortin system and of markers of white and brown adipocytes was evaluated along with mitochondrial biogenesis and cellular oxygen consumption. Metabolic evaluation of corticosterone-deficient or supplemented Crhr1-null (Crhr1−/−) mice and their wild-type controls was performed alo…

obesitycrf1Corticotropin-Releasing Hormonecrf2Endocrinology Diabetes and MetabolismIMPAIRED STRESS-RESPONSE[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionAdipocytes WhiteMedicine (miscellaneous)urocortinWhite adipose tissueMOUSEMicebrown adiposte tissue0302 clinical medicineBrowningUrocortinsUrocortin0303 health sciencesNutrition and Dietetics[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismParacrine mechanisms[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismImmunohistochemistryADIPOCYTESAdipocytes BrownADIPOSE-TISSUESKELETAL-MUSCLEhormones hormone substitutes and hormone antagonistsSignal TransductionEXPRESSIONmedicine.medical_specialtyendocrine systemTHERMOGENESISBiologycrfReceptors Corticotropin-Releasing Hormone03 medical and health scienceswhite adipose tissueInternal medicine3T3-L1 CellsmedicineAnimalsRNA MessengerGLUCOCORTICOIDS030304 developmental biologyENERGY HOMEOSTASISCorticotrophin releasing factoradipose plasticityPigments BiologicalUROCORTIN-II GENEQPEndocrinologyGene Expression Regulation[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgery
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Mitochondrial involvement in non-alcoholic steatohepatitis

2007

Non-alcoholic steatohepatitis (NASH) is an increasing recognized condition that may progress to end-stage liver disease. There are consistent evidences that mitochondrial dysfunction plays a central role in NASH whatever its origin. Mitochondria are the key controller of fatty acids removal and this is part of an intensive gene program that modifies hepatocytes to counteract the excessive fat storage. Mitochondrial dysfunction participates at different levels in NASH pathogenesis since it impairs fatty liver homeostasis and induces overproduction of ROS that in turn trigger lipid peroxidation, cytokines release and cell death. In this review we briefly recall the role of mitochondria in fat…

Mitochondrial DNAmedicine.medical_specialtyClinical BiochemistryBiologyMitochondrionModels BiologicalBiochemistryEnergy homeostasisAdenosine TriphosphateInternal medicinemedicineAnimalsHumansMolecular BiologyFatty liverGeneral MedicineTFAMLipid Metabolismmedicine.diseaseMitochondriaFatty LiverEndocrinologyMitochondrial respiratory chainMolecular MedicineSteatohepatitisSteatosisReactive Oxygen SpeciesMolecular Aspects of Medicine
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The role of the leptin in reproduction.

2006

Since its discovery in 1994, leptin has appeared to be a pleiotrophic hormone, governing energy homeostasis and affecting many tissues in the body. Numerous pieces of evidence have accumulated showing that leptin potentially plays an important role in the control of the reproductive function.This review presents the major concepts for the role of leptin in the modulation of reproductive function. As a marker of the nutritional status, leptin affects the hypothalamo-pituitary-gonadal axis, regulating gonadotrophin-releasing hormone and luteinising hormone secretion, and appears to be a permissive factor in the onset of the puberty. This protein and its receptor have been found in the reprodu…

Leptinmedicine.medical_specialtyLeptin receptorLeptinmedia_common.quotation_subjectReproductiondigestive oral and skin physiologyOvaryObstetrics and GynecologyBiologyEnergy homeostasisEndometriumEndocrinologyPregnancyInternal medicinemedicineHumansFemaleEmbryo ImplantationReproductionhormones hormone substitutes and hormone antagonistsHormonemedia_commonCurrent opinion in obstetricsgynecology
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Chatting Between the Brain and White Adipose Tissues

2012

Over the past decades, numerous papers have been published demonstrating the importance of the relationships between the brain and white adipose tissues in regard to body weight and metabolism regulation. Indeed the brain mainly via the sympathetic nervous system control body fat mass both by regulating adipocytes metabolism (lipolysis and lipogenesis), secretory activity (leptin and other adipokines) as well as development. In turn fat mass will send information to some brain areas via sensory nerves as well as via changes in metabolic and hormonal signals. Altogether these data are in support of a feedback loop between white adipose tissues and the brain. This crosstalk plays a major role…

medicine.medical_specialtyAutonomic nervous systemEndocrinologyInternal medicineLeptinLipogenesismedicineAdipokineLipolysisAdipose tissueWhite adipose tissueBiologyEnergy homeostasis
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Physiological and Nutritional Roles of PPAR across Species.

2013

There has been a tremendous amount of information produced on peroxisome proliferator-activated receptors (PPARs). The interest in PPARs was originally driven largely by their role in hypolipidemia and hepatocarcinogenesis, but it soon became evident that they played important roles in the metabolic syndrome and overall health of organisms including regeneration of tissues, differentiation, insulin signaling, overall lipid metabolism, and immune response (reviewed in [1–7]). From a nutritional standpoint, the PPARs are of extreme importance because of their ability to bind and be activated by long-chain fatty acids and their metabolites. Therefore, the PPARs are recognized as ideal candidat…

medicine.medical_specialtyArticle SubjectAnimal food[SDV.MHEP.PHY] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Peroxisome proliferator-activated receptorAdipose tissueContext (language use)White adipose tissueBiologyBioinformaticsEnergy homeostasis03 medical and health sciencesInternal medicineDrug Discoverymedicine[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular Biology[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Pharmacology (medical)[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biologylcsh:QH301-705.5[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyComputingMilieux_MISCELLANEOUS030304 developmental biology2. Zero hungerchemistry.chemical_classification[SDV.MHEP.EM] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism0303 health sciences[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]0402 animal and dairy scienceLipid metabolism04 agricultural and veterinary sciences[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism040201 dairy & animal scienceNutrigenomicsEndocrinologyEditoriallcsh:Biology (General)chemistry
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Functional Evaluation of THIQ, a Melanocortin 4 Receptor Agonist, in Models of Food Intake and Inflammation

2007

The central melanocortinergic system plays an important role in regulating different aspects of energy homeostasis and the immunomodulatory response. In the present study, we evaluated the in vivo activities of food intake suppression and anti-inflammatory activity of THIQ, which has been proposed to possess high and selective melanocortin-4 receptor agonistic activity in vitro. The results showed that THIQ (0.1, 0.3 and 1 nmol/rat, intracerebroventricularly) is less effective in reducing food intake and body weights of rats than the non-selective melanocortin receptor agonist melanotan II. Electron paramagnetic resonance measurements in mice brain tissue showed that THIQ at doses of 0.001 …

LipopolysaccharidesMaleAgonistmedicine.medical_specialtymedicine.drug_classAnti-Inflammatory AgentsBiologyNitric OxideToxicologyPeptides CyclicEnergy homeostasisEatingMiceMelanocortin receptorIn vivoTetrahydroisoquinolinesInternal medicinemedicineAnimalsRats WistarReceptorInjections IntraventricularInflammationPharmacologyMice Inbred ICRDose-Response Relationship DrugBody Weightdigestive oral and skin physiologyElectron Spin Resonance SpectroscopyBrainMelanotan IIGeneral MedicineTriazolesRatsMelanocortin 4 receptorDisease Models AnimalEndocrinologyalpha-MSHTHIQReceptor Melanocortin Type 4medicine.drugBasic & Clinical Pharmacology & Toxicology
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Postnatal Overfeeding in Rodents by Litter Size Reduction Induces Major Short- and Long-Term Pathophysiological Consequences

2013

Numerous studies have demonstrated that the early postnatal environment can influence body weight and energy homeostasis into adulthood. Rodents raised in small litters have been shown to be a useful experimental model to study the short- and long-term consequences of early overnutrition, which can lead to modifications not only in body weight but also of several metabolic features. Postnatal overfeeding (PNOF) induces early malprogramming of the hypothalamic system, inducing acquired persisting central leptin and insulin resistance and an increase in orexigenic signals. Visceral white adipose tissue, lipogenic activity, and inflammatory status are increased in PNOF rodents, while brown adi…

medicine.medical_specialtyLitter Sizemedia_common.quotation_subjectHypothalamusAppetiteMedicine (miscellaneous)White adipose tissueHyperphagiaBiologyEnergy homeostasisToxicology03 medical and health scienceschemistry.chemical_compoundOvernutrition0302 clinical medicineInsulin resistanceCorticosteroneInternal medicineBrown adipose tissuemedicineAnimalsHumansHormone metabolismObesityInfant Nutritional Physiological Phenomena030304 developmental biologymedia_common2. Zero hunger0303 health sciencesNutrition and DieteticsLeptinInfantAppetitemedicine.diseaseHormonesEndocrinologymedicine.anatomical_structureAdipose TissuechemistryCardiovascular DiseasesInsulin ResistanceEnergy Intake030217 neurology & neurosurgeryThe Journal of Nutrition
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The gliotransmitter ACBP controls feeding and energy homeostasis via the melanocortin system

2019

International audience; Glial cells have emerged as key players in the central control of energy balance and etiology of obesity. Astrocytes play a central role in neural communication via the release of gliotransmitters. Acyl-CoA binding protein (ACBP)-derived endozepines are secreted peptides that modulate the GABAA receptor. In the hypothalamus, ACBP is enriched in arcuate nucleus (ARC) astrocytes, ependymocytes and tanycytes. Central administration of the endozepine octadecaneuropeptide (ODN) reduces feeding and improves glucose tolerance, yet the contribution of endogenous ACBP in energy homeostasis is unknown. We demonstrated that ACBP deletion in GFAP+ astrocytes, but not in Nkx2.1-l…

0301 basic medicineMalePro-OpiomelanocortinGliotransmitter[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHyperphagiaEnergy homeostasisCell Lineneuroscience03 medical and health sciencesEatingMice0302 clinical medicineProopiomelanocortinCentral melanocortin systemmedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]AnimalsObesityComputingMilieux_MISCELLANEOUSDiazepam Binding InhibitorMice KnockoutNeuronsArc (protein)biologyChemistryGABAA receptorGeneral MedicineViral rescue[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolismCell biology030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisAstrocytesbiology.proteinFemale[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]MelanocortinEnergy Metabolismmetabolism[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyResearch Article
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Role of polysialic acid (PSA) in the control of food intake and body weight

2013

Hypothalamus plays a major role in the regulation of energy homeostasis by the presence of neural circuits controlling food intake. These circuits are plastic and can be rewired during adulthood. We hypothesized that synaptic plasticity can occur during physiological conditions. We have shown that synaptic contact on hypothalamic anorexigen POMC neurons are rewired in mouse upon high fat diet (HFD). This synaptic process is mandatory to adjust energy intake and requires the glycan PSA (polysialic acid). PSA promotes synaptic plasticity in the brain by the weakening of cell-to-cell interaction by addition on NCAM (neural cell adhesion molecule). We hypothesized that a defect in brain synapti…

[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]HypothalamusPrise alimentaireHoméostasie énergétiqueSynaptic plasticity[SDV.AEN] Life Sciences [q-bio]/Food and NutritionFood intakePlasticité synaptique[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Energy homeostasis[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]these[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Mitochondrial Fatty Acid β-Oxidation Inhibition Promotes Glucose Utilization and Protein Deposition through Energy Homeostasis Remodeling in Fish.

2020

BACKGROUND: Fish cannot use carbohydrate efficiently and instead utilize protein for energy supply, thus limiting dietary protein storage. Protein deposition is dependent on protein turnover balance, which correlates tightly with cellular energy homeostasis. Mitochondrial fatty acid β-oxidation (FAO) plays a crucial role in energy metabolism. However, the effect of remodeled energy homeostasis caused by inhibited mitochondrial FAO on protein deposition in fish has not been intensively studied. OBJECTIVES: This study aimed to identify the regulatory role of mitochondrial FAO in energy homeostasis maintenance and protein deposition by studying lipid, glucose, and protein metabolism in fish. M…

0301 basic medicineMaleProtein metabolismMedicine (miscellaneous)MitochondrionEnergy homeostasis03 medical and health scienceschemistry.chemical_compoundNile tilapia0302 clinical medicineAdjuvants ImmunologicmedicineAnimalsHomeostasisInsulinCarnitineProtein kinase ACells CulturedZebrafishNutrition and DieteticsbiologyCarnitine O-PalmitoyltransferaseChemistryFatty AcidsProtein turnoverProteinsMetabolismCichlidsDNACytochromes bbiology.organism_classificationMitochondria030104 developmental biologyGlucoseBiochemistryMutationHepatocytesNutrient Physiology Metabolism and Nutrient-Nutrient InteractionsEnergy MetabolismOxidation-Reduction030217 neurology & neurosurgerymedicine.drugMethylhydrazinesThe Journal of nutrition
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